Research Interest
Molecular and Cellular Virology - basic cellular, biochemical, and molecular biology of human influenza virus and astrovirus pathogenesis. Specific areas of interest include identification of cellular receptors for influenza A viruses and astrovirus, signal transduction pathways, the role of apoptosis and transforming growth factor-beta in viral pathogenesis, and the mechanism of astrovirus-induced diarrhea.
Training
1988 - B.S., University of Wisconsin-Platteville
1995 - Ph.D., University of Alabama at Birmingham
1995-97 - Postdoctoral Fellow, University of Wisconsin-Madison
Recent Publications NCBI PubMed search for "S. Schultz-Cherry"
Koci, M.D., Seal, B.S., and Schultz-Cherry, S. 2000. Molecular characterization of an avian astrovirus. J. Virol. 74:6173-6177.
Koci, M.D., Seal, B.S., and Schultz-Cherry, S. 2000. Development of an RT-PCR diagnostic test for an avian astrovirus. J. Virol Methods. 90:79-83.
Koci, M.D., and Schultz-Cherry, S. 2001. Avian Astroviruses. Avian Pathology. 31:213-229.
Koci, M.D., Larsen, D. Kelley, L., Brown, C., and Schultz-Cherry, S. 2002. Pathogenesis of a Novel Strain of Turkey Astrovirus: Viremia and Activation of TGF-beta. Submitted J. Virol.
Influenza
Schultz-Cherry, S., Dybing, J.K., Davis, N.L., Williamson, C., Suarez, D.L., Johnston, R. and Perdue, M.L. 2000. Influenza virus (A/HK/156/97) hemagglutinin expressed by an alphavirus replicon system protects chickens against lethal infection with Hong Kong-origin H5N1 viruses. Virology 278:55-59.
Schultz-Cherry, S., Dybdahl-Sissoko, N., Neumann, G., Kawaoka, Y., Krug, R., and Hinshaw, V.S. 2001. Influenza virus NS1 protein induces apoptosis in cultured cells. J. Virol. 75: 7875-7881.
Gantt, K.R., Schultz-Cherry, S., Rodriguez, N., Jeronimo, S.M.B., Nascimento, E.T., Goldman, T.L., Recker, T.J., Miller, M.A., Wilson, M.E. 2002. Activation of TGF-B by Leishmania chagasi: Importance for parasite survival in macrophages. Submitted J. Biol Chem.
Turpin, E., Tumpey, T., and Schultz-Cherry, S. 2002. Role for p53 in influenza virus-induced apoptosis. To be submitted J. Virol.